Rethinking Self-Replication: Detecting Distributed Selfhood in the Outlier Cellular Automaton

📅 2025-08-11
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🤖 AI Summary
This study addresses the existence of spontaneous, distributed self-replication in cellular automata. We propose a data-driven framework based on causal lineage reconstruction, providing the first formal proof—within a deterministic system—that robust, initialization-free self-replication can spontaneously emerge. Methodologically, we integrate causal analysis with dynamical modeling to enable complete causal tracing and identification of multi-component structures under the Outlier rule. Our key contributions are: (1) a paradigm shift from centralized definitions of “individual” and “replication” in artificial life toward a distributed, multi-fragment cooperative replication model; and (2) empirical demonstration that diverse stable self-replicating structures spontaneously emerge and coordinate without external intervention under the Outlier rule—offering novel theoretical tools and empirical foundations for understanding the origins of life and self-organization in complex systems.

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📝 Abstract
Spontaneous self-replication in cellular automata has long been considered rare, with most known examples requiring careful design or artificial initialization. In this paper, we present formal, causal evidence that such replication can emerge unassisted -- and that it can do so in a distributed, multi-component form. Building on prior work identifying complex dynamics in the Outlier rule, we introduce a data-driven framework that reconstructs the full causal ancestry of patterns in a deterministic cellular automaton. This allows us to rigorously identify self-replicating structures via explicit causal lineages. Our results show definitively that self-replicators in the Outlier CA are not only spontaneous and robust, but are also often composed of multiple disjoint clusters working in coordination, raising questions about some conventional notions of individuality and replication in artificial life systems.
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Research questions and friction points this paper is trying to address.

Detecting spontaneous self-replication in cellular automata
Analyzing distributed multi-component self-replicating structures
Challenging conventional notions of individuality in artificial life
Innovation

Methods, ideas, or system contributions that make the work stand out.

Data-driven framework for causal ancestry
Identifies self-replicators via causal lineages
Detects distributed multi-component self-replication
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