🤖 AI Summary
Children’s automatic speech recognition (ASR) performance lags significantly behind adults’ due to intertwined physiological (e.g., vocal tract morphology), cognitive (e.g., immature articulation), and extrinsic factors (e.g., limited vocabulary, environmental noise)—yet conventional analyses treat these in isolation. Method: This work pioneers the integration of causal structure discovery (PC/NOTEARS) and structural causal modeling (SCM) into ASR diagnostics for children, uncovering latent causal pathways such as “age → articulation → recognition errors,” quantifying direct and indirect effects, and conducting counterfactual intervention analysis via fine-tuning Whisper and Wav2Vec 2.0. Contribution/Results: Articulation proficiency is identified as the primary mediator of age-related ASR disparities. Fine-tuning effectively mitigates vocabulary constraints but yields marginal improvement on physiologically grounded acoustic mismatches. The framework demonstrates strong cross-model generalizability, advancing beyond reductionist, isolated-factor attribution paradigms in child ASR research.
📝 Abstract
Children's automatic speech recognition (ASR) often underperforms compared to that of adults due to a confluence of interdependent factors: physiological (e.g., smaller vocal tracts), cognitive (e.g., underdeveloped pronunciation), and extrinsic (e.g., vocabulary limitations, background noise). Existing analysis methods examine the impact of these factors in isolation, neglecting interdependencies-such as age affecting ASR accuracy both directly and indirectly via pronunciation skills. In this paper, we introduce a causal structure discovery to unravel these interdependent relationships among physiology, cognition, extrinsic factors, and ASR errors. Then, we employ causal quantification to measure each factor's impact on children's ASR. We extend the analysis to fine-tuned models to identify which factors are mitigated by fine-tuning and which remain largely unaffected. Experiments on Whisper and Wav2Vec2.0 demonstrate the generalizability of our findings across different ASR systems.